Understanding the Genetic Mechanisms Controlling Sow Longevity

dc.contributor.author Rothschild, Max
dc.contributor.author Mote, Benny
dc.contributor.author Stalder, Kenneth
dc.contributor.author Rothschild, Max
dc.contributor.author Stalder, Kenneth
dc.date 2018-08-25T22:38:09.000
dc.date.accessioned 2020-06-29T23:30:33Z
dc.date.available 2020-06-29T23:30:33Z
dc.date.copyright Mon Jan 01 00:00:00 UTC 2007
dc.date.issued 2007-01-01
dc.description.abstract <p>Sow longevity, or more accurately called sow productive life (SPL), has been a major focus point of concern in the swine industry for some time now. In addition to the animal and worker welfare concerns, industry leaders are realizing the substantial economical losses that occur when a farm has poor SPL in its commercial females. The economic benefits for increasing the average age of the females of reproductive age in the herd by one tenth of a parity are an additional $0.23 and $0.13 for every hog marketed in a farrow-tofinish and farrow-to-wean operations, respectively. Taken collectively, this sums up to over 15 million dollars in increased income in the US swine industry alone. Therefore even minor improvements to SPL can make an enormous impact of the financial bottom line for swine operations.</p> <p>Previous to this research, no studies have focused on the genes that play a pivotal role in SPL. Many researchers using model organisms such as nematodes, flies, and mice have shown a clear network of genes that play an integral role in increasing simple lifespan of these animals, primarily through a reduction in caloric intake or changes in the genes that mimic caloric restriction. These observations also hold true for human longevity studies. It is therefore our working hypothesis that these same genes may be involved in SPL. We realize that SPL and simple lifespan are not completely correlated. We are fully aware that in swine production thinner sows are typically culled earlier because they typically don’t have the body reserves to rely on during times of extreme nutrient requirements. We expect (and have encountered) alleles in these same genes from the model organisms that are associated with higher backfat to actually be the beneficial alleles for SPL. Our research has shown that there are several genes that can be selected for to improve the SPL of females at the commercial level.</p>
dc.identifier archive/lib.dr.iastate.edu/ans_air/vol653/iss1/57/
dc.identifier.articleid 1269
dc.identifier.contextkey 3327100
dc.identifier.doi https://doi.org/10.31274/ans_air-180814-942
dc.identifier.s3bucket isulib-bepress-aws-west
dc.identifier.submissionpath ans_air/vol653/iss1/57
dc.identifier.uri https://dr.lib.iastate.edu/handle/20.500.12876/8402
dc.language.iso en
dc.relation.ispartofseries Animal Science Research Reports
dc.source.bitstream archive/lib.dr.iastate.edu/ans_air/vol653/iss1/57/R2231.pdf|||Sat Jan 15 00:58:27 UTC 2022
dc.subject.disciplines Agriculture
dc.subject.disciplines Animal Sciences
dc.subject.keywords ASL R2231
dc.title Understanding the Genetic Mechanisms Controlling Sow Longevity
dc.type article
dc.type.genre swine
dspace.entity.type Publication
relation.isAuthorOfPublication a5915699-0c8a-411e-a601-1564d7090ab9
relation.isAuthorOfPublication 0b0a34a3-f123-4f94-a9cf-e730cb2183a6
relation.isJournalIssueOfPublication 70de9dc8-be88-49a2-93ca-08fadeb8478d
relation.isSeriesOfPublication 7f3839b7-b833-4418-a6fa-adda2b23950a
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